Tissue Repair and Fibrosis [electronic resource] : The Role of the Myofibroblast /

Some Historical and Philosophical Reflections on the Myofibroblast Concept -- Transcriptional Regulation of the Desmin and SM22 Genes in Vascular Smooth Muscle Cells -- Modulation of Myofibroblast and Smooth-Muscle Phenotypes in the Lung -- Role of Platelet-Derived Growth Factors in Angiogenesis and Alveogenesis -- Transforming Growth Factor-? Gene Transfer to the Lung Induces Myofibroblast Presence and Pulmonary Fibrosis -- Transforming Growth Factor-? Induction of ?-Smooth Muscle Actin is Dependent on the Deformability of the Collagen Matrix -- Signal Transduction Pathways Activated During Fibroblast Contraction of Collagen Matrices -- Extracellular Matrix, Integrins and Focal Adhesions -- What Is New in Mechanical Properties of Tissue-Engineered Organs -- Fibroblast - Cytokine - Extracellular Matrix Interactions in Wound Repair -- Myofibroblastic Differentiation and Extracellular Matrix Deposition in Early Stages of Cholestatic Fibrosis in Rat Liver -- Gene Therapy of Wounds with Growth Factors -- What’s New in Human Wound-Healing Myofibroblasts? -- The Myofibroblast in Neoplasia -- The Concept of Organizing Pneumonia -- Endothelial Repair in Atherogenesis -- Glomerulosclerosis: The Role of Interstitial Myofibroblasts in its Progression -- The Myofibroblast as an Inflammatory Cell in Pulmonary Fibrosis -- Cell-Cell and Cell-Matrix Interactions During Breast Cancer Progression -- Hepatocyte Growth Factor Secreted by Human Liver Myofibroblasts Increases Invasiveness of Hepatocellular Carcinoma Cells -- Interplay of Matrix and Myofibroblasts During Hepatic Fibrogenesis.

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Bibliographic Details
Main Authors: Desmoulière, Alexis. editor., Tuchweber, Beatriz. editor., SpringerLink (Online service)
Format: Texto biblioteca
Language:eng
Published: Berlin, Heidelberg : Springer Berlin Heidelberg : Imprint: Springer, 1999
Subjects:Medicine., Oncology., Pathology., Medicine & Public Health.,
Online Access:http://dx.doi.org/10.1007/978-3-642-58456-5
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