ON the pathogenesis of headache following TIA
Twelve out of 49 patients with single or multiple transient ischemic attacks (TIAs) had TIA-related headaches, mostly in close temporal relation to the ischemic onset. Headache predominated in patients taking vasodilators when TIA occurred or with orthostatic hypotension at the first clinical examination, but arterial hypertension or a personal history of migraine were not more frequent in patients with headache. The site of the pain did not correlate with the presumed territory of cerebral ischemia. Pain during TIA is conceivably due to an interaction between cerebral vessels and the surrounding nervous system. Blood vessels have a sturdy physiological role corcerning blood flow regulation, with receptors and signaling molecules potentialy involved with pain production. Reflex mechanisms should justify pain in other areas.
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Academia Brasileira de Neurologia - ABNEURO
1997
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oai:scielo:S0004-282X19970002000012010-11-10ON the pathogenesis of headache following TIAAndré,CharlesVincent,Maurice B. transient cerebral ischemia Cortisol headache neuroendocrine system serotonin trigeminovascular system Twelve out of 49 patients with single or multiple transient ischemic attacks (TIAs) had TIA-related headaches, mostly in close temporal relation to the ischemic onset. Headache predominated in patients taking vasodilators when TIA occurred or with orthostatic hypotension at the first clinical examination, but arterial hypertension or a personal history of migraine were not more frequent in patients with headache. The site of the pain did not correlate with the presumed territory of cerebral ischemia. Pain during TIA is conceivably due to an interaction between cerebral vessels and the surrounding nervous system. Blood vessels have a sturdy physiological role corcerning blood flow regulation, with receptors and signaling molecules potentialy involved with pain production. Reflex mechanisms should justify pain in other areas.info:eu-repo/semantics/openAccessAcademia Brasileira de Neurologia - ABNEUROArquivos de Neuro-Psiquiatria v.55 n.2 19971997-06-01info:eu-repo/semantics/articletext/htmlhttp://old.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X1997000200001en10.1590/S0004-282X1997000200001 |
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André,Charles Vincent,Maurice B. |
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André,Charles Vincent,Maurice B. ON the pathogenesis of headache following TIA |
| author_facet |
André,Charles Vincent,Maurice B. |
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André,Charles |
| title |
ON the pathogenesis of headache following TIA |
| title_short |
ON the pathogenesis of headache following TIA |
| title_full |
ON the pathogenesis of headache following TIA |
| title_fullStr |
ON the pathogenesis of headache following TIA |
| title_full_unstemmed |
ON the pathogenesis of headache following TIA |
| title_sort |
on the pathogenesis of headache following tia |
| description |
Twelve out of 49 patients with single or multiple transient ischemic attacks (TIAs) had TIA-related headaches, mostly in close temporal relation to the ischemic onset. Headache predominated in patients taking vasodilators when TIA occurred or with orthostatic hypotension at the first clinical examination, but arterial hypertension or a personal history of migraine were not more frequent in patients with headache. The site of the pain did not correlate with the presumed territory of cerebral ischemia. Pain during TIA is conceivably due to an interaction between cerebral vessels and the surrounding nervous system. Blood vessels have a sturdy physiological role corcerning blood flow regulation, with receptors and signaling molecules potentialy involved with pain production. Reflex mechanisms should justify pain in other areas. |
| publisher |
Academia Brasileira de Neurologia - ABNEURO |
| publishDate |
1997 |
| url |
http://old.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X1997000200001 |
| work_keys_str_mv |
AT andrecharles onthepathogenesisofheadachefollowingtia AT vincentmauriceb onthepathogenesisofheadachefollowingtia |
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