Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health

Global studies of the health impacts of fine particulate matter (PM2.5) have been based on particle mass, but there is evolving evidence indicating that adverse health effects can vary depending on the source and composition of PM2.5. This complicates the equitoxicity assumption commonly made regarding particle air pollution while raising the question as to which types of PM2.5 are most toxic. This report provides a comprehensive review of health effects of short- and long-term exposures to source-related components and trace constituents (specific markers of the individual sources) of fine particulate matter (PM2.5) in outdoor air pollution. The analysis evaluates the data and methods used, as well as the uncertainties in the underlying epidemiological studies, based on the relevant published literature. Most deaths attributed to outdoor PM2.5 air pollution (for example, by the Global Burden of Disease study) are caused by cardiovascular disease (CVD). The current evidence summarized in this report indicates that trace constituents from PM2.5 and PM2.5 mass from fossil-fuel combustion are among the greatest contributors to PM2.5 toxicity. Notably, PM2.5 from fossil-fuel combustion poses a larger cardiovascular disease risk per unit mass of PM2.5 than soil or biomass particles. Of the fossil-fuel combustion particles, coal- and traffic-related PM2.5 were found to be most consistently associated with cardiovascular mortality, especially due to ischemic heart disease (heart attacks) in both the short- and long-term exposure studies that were reviewed.

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Bibliographic Details
Main Authors: Thurston, George, Awe, Yewande, Ostro, Bart, Sanchez-Triana, Ernesto
Format: Report biblioteca
Language:English
Published: World Bank, Washington, DC 2021-09-03
Subjects:AIR POLLUTION, PARTICULATE MATTER, PM2.5, DAILY MORTALITY, HEALTH EFFECT,
Online Access:http://documents.worldbank.org/curated/undefined/810141630705865331/Are-All-Air-Pollution-Particles-Equal-How-Constituents-and-Sources-of-Fine-Air-Pollution-Particles-PM2-5-Affect-Health
http://hdl.handle.net/10986/36269
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spelling dig-okr-10986362692021-09-15T05:11:01Z Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health Thurston, George Awe, Yewande Ostro, Bart Sanchez-Triana, Ernesto AIR POLLUTION PARTICULATE MATTER PM2.5 DAILY MORTALITY HEALTH EFFECT Global studies of the health impacts of fine particulate matter (PM2.5) have been based on particle mass, but there is evolving evidence indicating that adverse health effects can vary depending on the source and composition of PM2.5. This complicates the equitoxicity assumption commonly made regarding particle air pollution while raising the question as to which types of PM2.5 are most toxic. This report provides a comprehensive review of health effects of short- and long-term exposures to source-related components and trace constituents (specific markers of the individual sources) of fine particulate matter (PM2.5) in outdoor air pollution. The analysis evaluates the data and methods used, as well as the uncertainties in the underlying epidemiological studies, based on the relevant published literature. Most deaths attributed to outdoor PM2.5 air pollution (for example, by the Global Burden of Disease study) are caused by cardiovascular disease (CVD). The current evidence summarized in this report indicates that trace constituents from PM2.5 and PM2.5 mass from fossil-fuel combustion are among the greatest contributors to PM2.5 toxicity. Notably, PM2.5 from fossil-fuel combustion poses a larger cardiovascular disease risk per unit mass of PM2.5 than soil or biomass particles. Of the fossil-fuel combustion particles, coal- and traffic-related PM2.5 were found to be most consistently associated with cardiovascular mortality, especially due to ischemic heart disease (heart attacks) in both the short- and long-term exposure studies that were reviewed. 2021-09-14T20:40:49Z 2021-09-14T20:40:49Z 2021-09-03 Report http://documents.worldbank.org/curated/undefined/810141630705865331/Are-All-Air-Pollution-Particles-Equal-How-Constituents-and-Sources-of-Fine-Air-Pollution-Particles-PM2-5-Affect-Health http://hdl.handle.net/10986/36269 English CC BY 3.0 IGO http://creativecommons.org/licenses/by/3.0/igo World Bank World Bank, Washington, DC Economic & Sector Work Economic & Sector Work :: Other Environmental Study
institution Banco Mundial
collection DSpace
country Estados Unidos
countrycode US
component Bibliográfico
access En linea
databasecode dig-okr
tag biblioteca
region America del Norte
libraryname Biblioteca del Banco Mundial
language English
topic AIR POLLUTION
PARTICULATE MATTER
PM2.5
DAILY MORTALITY
HEALTH EFFECT
AIR POLLUTION
PARTICULATE MATTER
PM2.5
DAILY MORTALITY
HEALTH EFFECT
spellingShingle AIR POLLUTION
PARTICULATE MATTER
PM2.5
DAILY MORTALITY
HEALTH EFFECT
AIR POLLUTION
PARTICULATE MATTER
PM2.5
DAILY MORTALITY
HEALTH EFFECT
Thurston, George
Awe, Yewande
Ostro, Bart
Sanchez-Triana, Ernesto
Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
description Global studies of the health impacts of fine particulate matter (PM2.5) have been based on particle mass, but there is evolving evidence indicating that adverse health effects can vary depending on the source and composition of PM2.5. This complicates the equitoxicity assumption commonly made regarding particle air pollution while raising the question as to which types of PM2.5 are most toxic. This report provides a comprehensive review of health effects of short- and long-term exposures to source-related components and trace constituents (specific markers of the individual sources) of fine particulate matter (PM2.5) in outdoor air pollution. The analysis evaluates the data and methods used, as well as the uncertainties in the underlying epidemiological studies, based on the relevant published literature. Most deaths attributed to outdoor PM2.5 air pollution (for example, by the Global Burden of Disease study) are caused by cardiovascular disease (CVD). The current evidence summarized in this report indicates that trace constituents from PM2.5 and PM2.5 mass from fossil-fuel combustion are among the greatest contributors to PM2.5 toxicity. Notably, PM2.5 from fossil-fuel combustion poses a larger cardiovascular disease risk per unit mass of PM2.5 than soil or biomass particles. Of the fossil-fuel combustion particles, coal- and traffic-related PM2.5 were found to be most consistently associated with cardiovascular mortality, especially due to ischemic heart disease (heart attacks) in both the short- and long-term exposure studies that were reviewed.
format Report
topic_facet AIR POLLUTION
PARTICULATE MATTER
PM2.5
DAILY MORTALITY
HEALTH EFFECT
author Thurston, George
Awe, Yewande
Ostro, Bart
Sanchez-Triana, Ernesto
author_facet Thurston, George
Awe, Yewande
Ostro, Bart
Sanchez-Triana, Ernesto
author_sort Thurston, George
title Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
title_short Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
title_full Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
title_fullStr Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
title_full_unstemmed Are All Air Pollution Particles Equal? How Constituents and Sources of Fine Air Pollution Particles (PM2.5) Affect Health
title_sort are all air pollution particles equal? how constituents and sources of fine air pollution particles (pm2.5) affect health
publisher World Bank, Washington, DC
publishDate 2021-09-03
url http://documents.worldbank.org/curated/undefined/810141630705865331/Are-All-Air-Pollution-Particles-Equal-How-Constituents-and-Sources-of-Fine-Air-Pollution-Particles-PM2-5-Affect-Health
http://hdl.handle.net/10986/36269
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