Cardiac Arrhythmias and Covid-19

Abstract Cardiovascular manifestations of COVID-19 include cardiac rhythm disturbances, whose mechanisms, incidence, and most common types are not well established in this population. Intense inflammatory response and metabolic activity contribute to recurrence of pre-existing arrhythmias, and other arrhythmias can occur due to myocardial injury, acute coronary insufficiency, and electrolyte disturbances. Brady- and tachyarrhythmias, as well as conduction disorders have been described. QT interval prolongation and fatal ventricular arrhythmias ( Torsades de Pointes ) may result from the pathological process or adverse effect of drugs (antiarrhythmics, chloroquine / hydroxychloroquine, azithromycin and antivirals). Patients with congenital heart disease and hemodynamic repercussions, patients with signs of heart failure, pulmonary hypertension, cyanosis, hypoxemia, and those who underwent heart transplantation and immunosuppression are at greater risk. In patients with implantable cardioverter-defibrillators (ICDs), the risk depends on the presence of structural heart disease. In the course of COVID-19, in-person assessment of these patients should be limited to high-risk situations, including syncope, worsening of heart failure and shock delivery by ICDs. Likewise, cardiac implantable electronic device implantation or replacement surgery should be limited to emergency and urgent cases, including symptomatic high-degree atrioventricular block, ICD for secondary prevention and pulse generator replacement due to battery drain.

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Main Authors: Fagundes,Alexsandro Alves, Melo,Sissy Lara de, Armaganijan,Luciana, Kuniyoshi,Ricardo, Moraes,Luis Gustavo Belo de, Borges,Vanessa Alves Guimarães, Scanavacca,Mauricio, Martinelli Filho,Martino, Teixeira,Ricardo Alkmim
Format: Digital revista
Language:English
Published: Sociedade Brasileira de Cardiologia 2021
Online Access:http://old.scielo.br/scielo.php?script=sci_arttext&pid=S2359-56472021005012201
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spelling oai:scielo:S2359-564720210050122012021-09-01Cardiac Arrhythmias and Covid-19Fagundes,Alexsandro AlvesMelo,Sissy Lara deArmaganijan,LucianaKuniyoshi,RicardoMoraes,Luis Gustavo Belo deBorges,Vanessa Alves GuimarãesScanavacca,MauricioMartinelli Filho,MartinoTeixeira,Ricardo Alkmim Coronavirus Infections Cardiac Arrhythmias Artificial Pacemaker Abstract Cardiovascular manifestations of COVID-19 include cardiac rhythm disturbances, whose mechanisms, incidence, and most common types are not well established in this population. Intense inflammatory response and metabolic activity contribute to recurrence of pre-existing arrhythmias, and other arrhythmias can occur due to myocardial injury, acute coronary insufficiency, and electrolyte disturbances. Brady- and tachyarrhythmias, as well as conduction disorders have been described. QT interval prolongation and fatal ventricular arrhythmias ( Torsades de Pointes ) may result from the pathological process or adverse effect of drugs (antiarrhythmics, chloroquine / hydroxychloroquine, azithromycin and antivirals). Patients with congenital heart disease and hemodynamic repercussions, patients with signs of heart failure, pulmonary hypertension, cyanosis, hypoxemia, and those who underwent heart transplantation and immunosuppression are at greater risk. In patients with implantable cardioverter-defibrillators (ICDs), the risk depends on the presence of structural heart disease. In the course of COVID-19, in-person assessment of these patients should be limited to high-risk situations, including syncope, worsening of heart failure and shock delivery by ICDs. Likewise, cardiac implantable electronic device implantation or replacement surgery should be limited to emergency and urgent cases, including symptomatic high-degree atrioventricular block, ICD for secondary prevention and pulse generator replacement due to battery drain.info:eu-repo/semantics/openAccessSociedade Brasileira de CardiologiaInternational Journal of Cardiovascular Sciences n.ahead 20212021-01-01info:eu-repo/semantics/articletext/htmlhttp://old.scielo.br/scielo.php?script=sci_arttext&pid=S2359-56472021005012201en10.36660/ijcs.20200123
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language English
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author Fagundes,Alexsandro Alves
Melo,Sissy Lara de
Armaganijan,Luciana
Kuniyoshi,Ricardo
Moraes,Luis Gustavo Belo de
Borges,Vanessa Alves Guimarães
Scanavacca,Mauricio
Martinelli Filho,Martino
Teixeira,Ricardo Alkmim
spellingShingle Fagundes,Alexsandro Alves
Melo,Sissy Lara de
Armaganijan,Luciana
Kuniyoshi,Ricardo
Moraes,Luis Gustavo Belo de
Borges,Vanessa Alves Guimarães
Scanavacca,Mauricio
Martinelli Filho,Martino
Teixeira,Ricardo Alkmim
Cardiac Arrhythmias and Covid-19
author_facet Fagundes,Alexsandro Alves
Melo,Sissy Lara de
Armaganijan,Luciana
Kuniyoshi,Ricardo
Moraes,Luis Gustavo Belo de
Borges,Vanessa Alves Guimarães
Scanavacca,Mauricio
Martinelli Filho,Martino
Teixeira,Ricardo Alkmim
author_sort Fagundes,Alexsandro Alves
title Cardiac Arrhythmias and Covid-19
title_short Cardiac Arrhythmias and Covid-19
title_full Cardiac Arrhythmias and Covid-19
title_fullStr Cardiac Arrhythmias and Covid-19
title_full_unstemmed Cardiac Arrhythmias and Covid-19
title_sort cardiac arrhythmias and covid-19
description Abstract Cardiovascular manifestations of COVID-19 include cardiac rhythm disturbances, whose mechanisms, incidence, and most common types are not well established in this population. Intense inflammatory response and metabolic activity contribute to recurrence of pre-existing arrhythmias, and other arrhythmias can occur due to myocardial injury, acute coronary insufficiency, and electrolyte disturbances. Brady- and tachyarrhythmias, as well as conduction disorders have been described. QT interval prolongation and fatal ventricular arrhythmias ( Torsades de Pointes ) may result from the pathological process or adverse effect of drugs (antiarrhythmics, chloroquine / hydroxychloroquine, azithromycin and antivirals). Patients with congenital heart disease and hemodynamic repercussions, patients with signs of heart failure, pulmonary hypertension, cyanosis, hypoxemia, and those who underwent heart transplantation and immunosuppression are at greater risk. In patients with implantable cardioverter-defibrillators (ICDs), the risk depends on the presence of structural heart disease. In the course of COVID-19, in-person assessment of these patients should be limited to high-risk situations, including syncope, worsening of heart failure and shock delivery by ICDs. Likewise, cardiac implantable electronic device implantation or replacement surgery should be limited to emergency and urgent cases, including symptomatic high-degree atrioventricular block, ICD for secondary prevention and pulse generator replacement due to battery drain.
publisher Sociedade Brasileira de Cardiologia
publishDate 2021
url http://old.scielo.br/scielo.php?script=sci_arttext&pid=S2359-56472021005012201
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