Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] /
In this book on diabetes mellitus both the pathogenesis and treat ment of the disease will be discussed. Pathogenetic studies have led to the distinction between type I and type II diabetes. In type I hyperglycaemia is due predominantly to insulin deficiency; in type II insulin resistance is more important. Three pathogenetic factors are thought to play a role in the etiol ogy of type I diabetes: heredity, viral infections and immunity. There may be a relationship between these three aspects since genetic pre disposition changes the susceptibility to viral infections and/or the immunological defence against these infections. Insulitis ensues. Auto immune processes initiated by this chain of events may contribute to the destruction of the B cells in the islets of Langerhans and diabetes -may eventually follow. This means that at the time of the sudden out break of the disease, the destructive process has already been active for years.
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Format: | Texto biblioteca |
Language: | eng |
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Dordrecht : Springer Netherlands,
1986
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Subjects: | Medicine., Internal medicine., Diabetes., Medicine & Public Health., Internal Medicine., |
Online Access: | http://dx.doi.org/10.1007/978-94-009-4301-8 |
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KOHA-OAI-TEST:2318172018-07-31T00:14:53ZPathogenesis and Treatment of Diabetes Mellitus [electronic resource] / Radder, J. K. editor. Lemkes, H. H. P. J. editor. Krans, H. M. J. editor. SpringerLink (Online service) textDordrecht : Springer Netherlands,1986.engIn this book on diabetes mellitus both the pathogenesis and treat ment of the disease will be discussed. Pathogenetic studies have led to the distinction between type I and type II diabetes. In type I hyperglycaemia is due predominantly to insulin deficiency; in type II insulin resistance is more important. Three pathogenetic factors are thought to play a role in the etiol ogy of type I diabetes: heredity, viral infections and immunity. There may be a relationship between these three aspects since genetic pre disposition changes the susceptibility to viral infections and/or the immunological defence against these infections. Insulitis ensues. Auto immune processes initiated by this chain of events may contribute to the destruction of the B cells in the islets of Langerhans and diabetes -may eventually follow. This means that at the time of the sudden out break of the disease, the destructive process has already been active for years.1.: Pathogenesis of Diabetes Mellitus -- Section 1.1.: Type I Diabetes Mellitus -- 1.1.1.: Introduction: Type I Diabetes Mellitus -- 1.1.2.: Autoimmune pathogenesis of viral diabetes -- 1.1.3.:Immunological features of insulin-dependent diabetes mellitus -- 1.1.4.:Genetic counselling in Diabetes Mellitus -- Section 1.2.: Type II Diabetes Mellitus -- 1.2.1.: Introduction: Pathogenesis of type II Diabetes Mellitus -- 1.2.2.:Insulin secretion in noninsulin-dependent Diabetes Mellitus -- 1.2.3.: Insulin antagonism -- 1.2.4.: The contribution of the central nervous system to the regulation of insulin release and the stabilization of blood glucose -- 1.2.5.: Cellular insulin resistance -- 2.: Treatment of Diabetes Mellitus -- Section 2.1.: Diet and hypoglycaemic agents -- 2.1.1.: Introduction: Diet and hypoglycaemic agents -- 2.1.2.: Diet and Diabetes -- 2.1.3.: Oral hypoglycaemic drugs -- Section 2.2.: Insulin -- 2.2.1.: Introduction: Insulin treatment -- 2.2.2.: Hyperinsulinaemia -- 2.2.3.: Insulin: The physiological basis of its administration -- 2.2.4.: Diabetes Technology: From the pump to the microprocessor -- Section 2.3.: Pancreas transplantation -- 2.3.1.: Introduction: Pancreas transplantation -- 2.3.2.: Current state of pancreas transplantation -- Index of subjects.In this book on diabetes mellitus both the pathogenesis and treat ment of the disease will be discussed. Pathogenetic studies have led to the distinction between type I and type II diabetes. In type I hyperglycaemia is due predominantly to insulin deficiency; in type II insulin resistance is more important. Three pathogenetic factors are thought to play a role in the etiol ogy of type I diabetes: heredity, viral infections and immunity. There may be a relationship between these three aspects since genetic pre disposition changes the susceptibility to viral infections and/or the immunological defence against these infections. Insulitis ensues. Auto immune processes initiated by this chain of events may contribute to the destruction of the B cells in the islets of Langerhans and diabetes -may eventually follow. This means that at the time of the sudden out break of the disease, the destructive process has already been active for years.Medicine.Internal medicine.Diabetes.Medicine & Public Health.Internal Medicine.Diabetes.Springer eBookshttp://dx.doi.org/10.1007/978-94-009-4301-8URN:ISBN:9789400943018 |
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Medicine. Internal medicine. Diabetes. Medicine & Public Health. Internal Medicine. Diabetes. Medicine. Internal medicine. Diabetes. Medicine & Public Health. Internal Medicine. Diabetes. |
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Medicine. Internal medicine. Diabetes. Medicine & Public Health. Internal Medicine. Diabetes. Medicine. Internal medicine. Diabetes. Medicine & Public Health. Internal Medicine. Diabetes. Radder, J. K. editor. Lemkes, H. H. P. J. editor. Krans, H. M. J. editor. SpringerLink (Online service) Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
description |
In this book on diabetes mellitus both the pathogenesis and treat ment of the disease will be discussed. Pathogenetic studies have led to the distinction between type I and type II diabetes. In type I hyperglycaemia is due predominantly to insulin deficiency; in type II insulin resistance is more important. Three pathogenetic factors are thought to play a role in the etiol ogy of type I diabetes: heredity, viral infections and immunity. There may be a relationship between these three aspects since genetic pre disposition changes the susceptibility to viral infections and/or the immunological defence against these infections. Insulitis ensues. Auto immune processes initiated by this chain of events may contribute to the destruction of the B cells in the islets of Langerhans and diabetes -may eventually follow. This means that at the time of the sudden out break of the disease, the destructive process has already been active for years. |
format |
Texto |
topic_facet |
Medicine. Internal medicine. Diabetes. Medicine & Public Health. Internal Medicine. Diabetes. |
author |
Radder, J. K. editor. Lemkes, H. H. P. J. editor. Krans, H. M. J. editor. SpringerLink (Online service) |
author_facet |
Radder, J. K. editor. Lemkes, H. H. P. J. editor. Krans, H. M. J. editor. SpringerLink (Online service) |
author_sort |
Radder, J. K. editor. |
title |
Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
title_short |
Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
title_full |
Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
title_fullStr |
Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
title_full_unstemmed |
Pathogenesis and Treatment of Diabetes Mellitus [electronic resource] / |
title_sort |
pathogenesis and treatment of diabetes mellitus [electronic resource] / |
publisher |
Dordrecht : Springer Netherlands, |
publishDate |
1986 |
url |
http://dx.doi.org/10.1007/978-94-009-4301-8 |
work_keys_str_mv |
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